12 12 A1.1 Start 13,216,973 End 13,249,213 pattern Molecular function. Cellular component. Biological process. Sources: / Species Human Mouse RefSeq (mRNA) RefSeq (protein) Location (UCSC) search ATP-dependent RNA helicase DDX1 is an that in humans is encoded by the DDX1. DEAD box proteins, characterized by the conserved motif Asp-Glu-Ala-Asp (DEAD), are putative RNA helicases. They are implicated in a number of cellular processes involving alteration of RNA secondary structure such as translation initiation, nuclear and mitochondrial splicing, and ribosome and spliceosome assembly.
Based on their distribution patterns, some members of this family are believed to be involved in embryogenesis, spermatogenesis, and cellular growth and division. This gene encodes a DEAD box protein of unknown function. It shows high transcription levels in 2 retinoblastoma cell lines and in tissues of neuroectodermal origin.
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Interactions DDX1 has been shown to with. References.
Corelap layout software. Link of the software https://drive.google.com/file/d/1zOvP7Ovyiu0DIeY3MP8DFlxX3t0ECAh8/view?usp=sharing. Computerized Layout Planning. » E.g., CORELAP. INEN 416 Facility Location, Layout, and Material Handling 10/9/2004 Texas A&M Industrial Engineering 9 INEN 416 124.
Contents. Function proteins, characterized by the conserved motif Asp-Glu-Ala-Asp (DEAD), are putative.
They are implicated in a number of cellular processes involving alteration of RNA secondary structure such as translation initiation, nuclear and mitochondrial splicing, and and assembly. Based on their distribution patterns, some members of this family are believed to be involved in, and cellular growth and division. This gene encodes a DEAD box protein, which has an activity and is a component of the (SMN) complex. This DEAD box protein is one of the gem-associated proteins (GEMIN3) that interacts directly with SMN. SMN is the spinal muscular atrophy gene product, and may play a catalytic role in the function of the SMN complex on RNPs. Biological Implication Previous research has revealed that DDX20 may act as a tumor suppressor in and as a promoter in.
DDX20 deficiency enhances activity by impairing the NF-κB-suppressive action of, and suggest that dysregulation of the microRNA machinery components may also be involved in pathogenesis in various human diseases. Such as miRNA-140 which acts as a liver tumor suppressor, and due to a deficiency of DDX20, miRNA-140 function gets impair, this subsequent functional impairment of miRNAs could lead to hepatocarcinogenesis.Similarly, DDX20 may promote the progression of (PCa) through the NF-κB pathway. In a clinical based study it has been observed that positive DP103/NF-κB feedback loop promotes constitutive NF-κB activation in invasive breast cancers and activation of this pathway is linked to cancer progression and the acquisition of resistance. It makes DP103 has potential as a therapeutic target for breast cancer treatment. Interactions DDX20 has been shown to with.